Ferret Diet and Nutrition Guide

Quick Q&A

Question: What is the most important nutritional requirement for a ferret? Answer: Ferrets are obligate carnivores requiring a diet exceptionally high in animal-based protein (30-40% minimum) and fat (15-30%) with very low carbohydrates. Their digestive systems are designed to process whole prey, and they cannot efficiently digest plant-based ingredients. A diet mimicking their natural prey is essential for health.

Question: Can ferrets eat dog or cat food? Answer: No. Ferrets have unique nutritional needs that differ significantly from dogs and cats. Dog food lacks sufficient protein and fat, while cat food, though closer, still does not meet the specific amino acid and fatty acid profiles required by ferrets. Feeding inappropriate diets can lead to malnutrition, obesity, and disease.

Question: What foods are toxic to ferrets? Answer: Many human foods are dangerous for ferrets, including chocolate, caffeine, alcohol, grapes, raisins, onions, garlic, xylitol (artificial sweetener), and dairy products. Fruits and vegetables should also be avoided as they are high in carbohydrates and low in nutritional value for obligate carnivores.

Introduction

The domestic ferret (Mustela putorius furo) is a small carnivore of the family Mustelidae, sharing a common ancestry with wild polecats and the endangered black-footed ferret (Mustela nigripes) [5]. As obligate carnivores, ferrets possess a short, simple gastrointestinal tract designed for the rapid digestion of animal tissues, lacking the cecum and enzymatic capacity to process plant matter efficiently [8, 11]. Their metabolic and physiological systems, including the endocrine, respiratory, and gastrointestinal systems, are adapted to a diet of whole prey, primarily small mammals [5, 6].

Inappropriate nutrition is a leading cause of morbidity and mortality in pet ferrets, contributing to conditions such as insulinoma, adrenal disease, gastrointestinal disorders, and obesity [8, 36]. This comprehensive guide, grounded in current veterinary science and clinical guidelines from the Merck Veterinary Manual and VCA Animal Hospitals, provides an exhaustive overview of ferret dietary requirements, feeding strategies, and nutritional management.

The Obligate Carnivore: Metabolic and Digestive Foundations

Ferrets are strict carnivores, meaning their diet must consist of animal-based tissues. This is reflected in their anatomy and physiology:

• Gastrointestinal Tract: Ferrets have a short gastrointestinal tract (approximately 5-7 times their body length) with a simple stomach and no functional cecum. This design is optimized for the rapid digestion of proteins and fats, not for fermenting fibrous plant material [5, 13].
• Dentition: Their dental formula (I 3/3, C 1/1, P 3/3, M 1/2) includes sharp, pointed teeth for shearing meat, not grinding plant matter. Studies on cranial morphology in black-footed ferrets have shown that captive diets can significantly alter skull shape compared to wild counterparts consuming whole prey, underscoring the impact of diet on physical development [10].
• Metabolic Pathways: Ferrets have a high metabolic rate and require a diet rich in energy-dense nutrients. They have a limited ability to metabolize carbohydrates. High carbohydrate intake can lead to pancreatic beta-cell hyperplasia and predispose ferrets to insulinoma, a common endocrine neoplasia in this species [8, 36]. Research on dietary shifts in pandas (also Carnivora) reveals that carnivores lack the genetic and epigenetic adaptations for digesting plant-based diets [11].
• Unique Glucuronidation: Ferrets, like cats, exhibit slow glucuronidation of certain compounds, including soy isoflavones and some drugs [38, 39]. This is an evolutionary consequence of a carnivorous diet low in plant-based toxins. This has implications for feeding plant-based ingredients and for the metabolism of certain medications.

Core Nutritional Requirements

According to the Merck Veterinary Manual and current veterinary consensus, a ferret's diet must meet specific macronutrient and micronutrient targets [8, 13].

Protein

Protein is the most critical nutrient. Ferrets require a minimum of 30-40% crude protein on a dry matter basis, with a preference for animal-based sources. The protein must be highly digestible and provide a complete amino acid profile, particularly taurine and arginine, which are essential for cardiac function, vision, and reproduction [8]. Plant-based proteins are poorly utilized and can create amino acid imbalances.

Fat

Fat is the primary energy source for ferrets. A minimum of 15-30% crude fat on a dry matter basis is recommended. High fat levels are not only acceptable but necessary to meet their energy demands. Essential fatty acids, such as linoleic acid and arachidonic acid (found only in animal fats), are crucial for skin health, coat quality, and immune function [8, 12]. The optimal protein-to-fat ratio is approximately 2:1 [8].

Carbohydrates

Ferrets have no dietary requirement for carbohydrates. Their natural prey diet is extremely low in carbs. Commercial diets should have minimal carbohydrate content, ideally less than 10-15% on a dry matter basis. High carbohydrate diets are strongly associated with the development of insulinoma and obesity [8, 36].

Fiber

Fiber is not a required nutrient for ferrets and should be kept low (less than 3-5%). Their digestive system is not adapted to handle it, and excessive fiber can interfere with nutrient absorption.

Vitamins and Minerals

Ferrets require a balanced supply of vitamins and minerals, which are best provided through a diet of whole animal tissues. Key considerations include:

• Taurine: An essential amino acid for ferrets, crucial for heart health and vision. It is found almost exclusively in animal tissues.
• Vitamin E: A fat-soluble antioxidant. Supplementation has been shown to positively influence reproductive hormones (fecal androgen metabolites) in male black-footed ferrets during the breeding season [16].
• Calcium and Phosphorus: A proper calcium-to-phosphorus ratio (approximately 1.2:1 to 1.5:1) is critical for bone health. Whole prey provides an ideal balance, whereas muscle meat alone is deficient in calcium.

Feeding Strategies: Kibble vs. Raw vs. Whole Prey

There is ongoing debate among owners and veterinarians regarding the optimal feeding method for ferrets. Each approach has benefits and risks.

Commercial Kibble

High-quality, commercial ferret kibble is the most convenient and commonly used option.

• Advantages: Balanced nutrition, long shelf life, convenient, and reduces the risk of bacterial contamination compared to raw diets.
• Disadvantages: Often contains higher carbohydrate levels than ideal, may include plant-based proteins and fillers, and can be highly processed. The heat processing of kibble can degrade some heat-sensitive nutrients.
• Selection Criteria: Choose a kibble specifically formulated for ferrets. The first three ingredients should be named animal protein sources (e.g., chicken meal, lamb meal, salmon). The crude protein should be 35-40% or higher, and crude fat 18-22% or higher. Avoid foods with grains, corn, wheat, soy, or high levels of peas or potatoes. A 2024 update on ferret nutrition emphasizes that a combination of dietary formats is often beneficial, as no single commercial diet perfectly meets all physiological needs [8].

Raw Feeding

Raw feeding involves providing uncooked meat, bones, and organs. This can be in the form of commercially prepared raw frozen diets or homemade recipes.

• Advantages: Closer to a natural diet, higher moisture content, lower carbohydrates, and better dental health from chewing bones.
• Disadvantages: Risk of bacterial contamination (e.g., Salmonella, E. coli, Campylobacter), nutritional imbalance if not properly formulated, and potential for zoonotic disease transmission. A 2024 study highlighted the risk of Highly Pathogenic Avian Influenza (H5N1) transmission to pet ferrets, advising against feeding fresh or frozen poultry to reduce this risk [2]. Another study noted that ground meat-based diets in captive black-footed ferrets were associated with higher levels of spore-forming bacteria in feces compared to whole-prey diets [15].
• Veterinary Guidance: If choosing a raw diet, it is crucial to use a commercially prepared, nutritionally complete raw food from a reputable manufacturer or to work with a veterinary nutritionist to formulate a balanced homemade diet.

Whole Prey

Whole prey feeding (e.g., whole mice, rats, chicks, quail) is considered the most biologically appropriate diet for ferrets.

• Advantages: Most natural, provides complete and balanced nutrition (including bones, organs, fur/feathers), promotes dental health, and supports natural foraging and feeding behaviors. Studies in black-footed ferrets have shown that a whole-prey diet can reduce stress (fecal glucocorticoid metabolites) and improve reproductive outcomes compared to ground meat diets [16].
• Disadvantages: Can be unappealing to some owners, requires sourcing and storage of frozen prey, and carries a risk of parasitic or bacterial transmission if prey is not from a reputable source.
• Clinical Evidence: Research on ex situ black-footed ferrets indicates that diet is a major environmental factor influencing fertility and epigenetic profiles [1, 6]. The natural diet of prairie dogs is critical for their reproductive success. While not practical for pet ferrets, this underscores the profound impact of diet on health and reproduction.

Dangerous Foods and Toxic Substances

Several common foods are highly dangerous or toxic to ferrets and must be strictly avoided [8].

• Dairy Products: Ferrets are lactose intolerant. Milk, cheese, and ice cream can cause severe diarrhoea and gastrointestinal upset.
• Fruits and Vegetables: These are high in simple carbohydrates and fiber, which ferrets cannot digest. They offer no nutritional benefit and can contribute to obesity and insulinoma. Fruits are highly palatable but should be avoided [8].
• Chocolate and Caffeine: Contain methylxanthines, which are toxic to ferrets and can cause vomiting, diarrhoea, hyperactivity, seizures, and death.
• Xylitol: An artificial sweetener found in sugar-free gum, candy, and some peanut butters. It causes a rapid, life-threatening drop in blood sugar and liver failure.
• Grapes and Raisins: Can cause acute kidney failure in ferrets.
• Onions and Garlic: Can damage red blood cells and cause hemolytic anemia.
• Alcohol: Extremely toxic, causing severe central nervous system depression and liver damage.
• Raw or Undercooked Poultry: As noted above, due to the risk of H5N1 avian influenza transmission, fresh or frozen poultry should be excluded from the diet [2].

Treats

Treats should be used sparingly and should be species-appropriate.

• Safe Treats: Small pieces of cooked, unseasoned meat (chicken, turkey, beef), freeze-dried meat treats (e.g., chicken hearts, liver), or commercial ferret treats that are high in protein and low in carbohydrates.
• Treats to Avoid: Commercial treats high in sugar, starch, or grains (e.g., "ferret yogies," fruit-flavored treats). These are detrimental to health.
• Frequency: Treats should constitute no more than 5-10% of the total daily caloric intake.

Nutritional Management of Common Diseases

Proper nutrition is a cornerstone of managing several common ferret diseases.

Insulinoma

Insulinoma, a pancreatic beta-cell tumor, is extremely common in ferrets over 3-4 years of age [36]. It is strongly linked to high-carbohydrate diets.

• Dietary Management: The primary goal is to stabilize blood glucose levels. This involves feeding a high-protein, high-fat, very low-carbohydrate diet. Frequent, small meals (every 4-6 hours) are often recommended to prevent hypoglycemia. A raw or whole-prey diet is ideal. If using kibble, choose a low-carbohydrate option. In some cases, a small amount of a high-protein supplement may be recommended between meals.

Adrenal Disease (Hyperadrenocorticism)

Adrenal disease is another common endocrinopathy in ferrets, often associated with early neutering and photoperiod [36]. While not directly caused by diet, nutrition plays a supportive role.

• Dietary Management: A high-quality, species-appropriate diet supports overall health and immune function. Supplementing with melatonin (under veterinary guidance) can help manage clinical signs. Ensuring adequate intake of omega-3 fatty acids may have anti-inflammatory benefits.

Obesity

Obesity is a growing problem in pet ferrets, often due to overfeeding of high-carbohydrate kibble and lack of exercise.

• Dietary Management: Feed a measured amount of a low-carbohydrate, high-protein diet. Avoid free-choice feeding. Increase exercise and enrichment. A study on diet-induced obesity in ferrets showed that obesity alters the lung microenvironment and increases susceptibility to severe influenza, highlighting the systemic health risks of obesity [4, 7].

Gastrointestinal Disorders

Gastroenteritis is a common cause of morbidity in ferrets [15]. Diet plays a key role in both prevention and management.

• Dietary Management: Feeding a whole-prey or high-quality raw diet is associated with lower levels of potentially pathogenic spore-forming bacteria in the feces compared to ground meat diets [15]. For ferrets with diarrhoea or vomiting, a bland diet of cooked chicken and bone broth (low sodium) may be recommended temporarily. Probiotics may be beneficial in some cases, though evidence is limited.

Feeding Schedule and Water

• Feeding Frequency: Ferrets have a fast metabolic rate and a short gastrointestinal transit time (approximately 3-4 hours). They should have access to food at all times (ad libitum feeding) or be fed multiple small meals throughout the day (at least 4-6 times). This is especially important for preventing hypoglycemia.
• Water: Fresh, clean water must be available at all times. Ferrets can be messy drinkers, so heavy bowls or water bottles are recommended. Water intake should be monitored, as changes can indicate illness.

Regional Considerations

• United States and Canada: The AVMA and AAHA do not have specific ferret nutrition guidelines, but the Merck Veterinary Manual and VCA Animal Hospitals provide authoritative advice. Owners should be aware of the risk of H5N1 from raw poultry [2].
• Europe: The FVE and EFSA provide general guidance on exotic pet welfare. European owners should be aware of regional differences in commercial food availability and veterinary expertise.
• Australia: Ferret ownership is regulated in some states. The AVA provides general guidance. Owners must ensure their ferret's diet complies with local biosecurity laws, particularly regarding raw meat.

Conclusion

Proper nutrition is the single most important factor in maintaining the health and longevity of a pet ferret. As obligate carnivores, their diet must be centered on high-quality animal protein and fat, with minimal carbohydrates. Owners should choose a feeding strategy that is safe, balanced, and practical, whether it be high-quality kibble, a commercially prepared raw diet, or whole prey. Avoiding dangerous foods and treats, and working closely with a veterinarian experienced in ferret medicine, will help prevent common diet-related diseases such as insulinoma, obesity, and gastrointestinal disorders. By understanding and meeting the unique nutritional needs of these fascinating animals, owners can ensure a long, healthy, and active life for their ferrets.

References

[1] DeCandia AL, Tennenbaum SR, Santymire R, et al. Ex situ reared black-footed ferrets exhibit altered sperm DNA methylation. J Hered. 2025. [2] Golke A, Jańczak D, Szaluś-Jordanow O, et al. Natural Infection with Highly Pathogenic Avian Influenza A/H5N1 Virus in Pet Ferrets. Viruses. 2024. [3] Knoll M, Honce R, Meliopoulos V, et al. Host obesity impacts genetic variation in influenza A viral populations. J Virol. 2024. [4] Meliopoulos V, Honce R, Livingston B, et al. Diet-induced obesity affects influenza disease severity and transmission dynamics in ferrets. Sci Adv. 2024. [5] Pramod RK, Atul PK, Pandey M, et al. Care, management, and use of ferrets in biomedical research. Lab Anim Res. 2024. [6] Ali N, Amelkina O, Santymire RM, et al. Semen proteome and transcriptome of the endangered black-footed ferret (Mustela nigripes) show association with the environment and fertility outcome. Sci Rep. 2024. [7] Meliopoulos V, Honce R, Livingston B, et al. Diet-induced obesity impacts influenza disease severity and transmission dynamics in ferrets. bioRxiv. 2023. [8] Iske C. An Update on Key Nutritional Factors in Ferret Nutrition. Vet Clin North Am Exot Anim Pract. 2024. [9] Eads DA, Tretten TN, Hughes JP, et al. LETHAL EFFECTS ON FLEA LARVAE OF FIPRONIL IN HOST FECES: POTENTIAL BENEFITS FOR PLAGUE MITIGATION. J Wildl Dis. 2023. [10] Antonelli T, Leischner CL, Hartstone-Rose A. The Cranial Morphology of the Black-Footed Ferret: A Comparison of Wild and Captive Specimens. Animals (Basel). 2022. [11] Li L, Shen F, Jie X, et al. Comparative Transcriptomics and Methylomics Reveal Adaptive Responses of Digestive and Metabolic Genes to Dietary Shift in Giant and Red Pandas. Genes (Basel). 2022. [12] Hryciw DH, Jackson CA, Shrestha N, et al. Role for animal models in understanding essential fatty acid deficiency in cystic fibrosis. Cell Mol Life Sci. 2021. [13] Bullen LE. Nutrition for Pocket Pets (Ferrets, Rabbits, and Rodents). Vet Clin North Am Small Anim Pract. 2021. [14] Mustra Rakic J, Liu C, Veeramachaneni S, et al. Dietary lycopene attenuates cigarette smoke-promoted nonalcoholic steatohepatitis by preventing suppression of antioxidant enzymes in ferrets. J Nutr Biochem. 2021. [15] Ferris RL, Stacy N, Stein AB, et al. COMPARISON OF FECAL CYTOLOGY AND PRESENCE OF CLOSTRIDIUM PERFRINGENS ENTEROTOXIN IN CAPTIVE BLACK-FOOTED FERRETS (MUSTELA NIGRIPES) BASED ON DIET AND FECAL QUALITY. J Zoo Wildl Med. 2021. [16] Santymire RM, Lavin SR, Branvold-Faber H, et al. Influence of vitamin E and carcass feeding supplementation on fecal glucocorticoid and androgen metabolites in male black-footed ferrets (Mustela nigripes). PLoS One. 2020. [17] Xiang Z, Zhu H, Yang B, et al. A glance at the gut microbiota of five experimental animal species through fecal samples. Sci Rep. 2020. [18] Salas H, Castillejos L, Faturi C, et al. Effects of replacing canola meal with camelina expeller on intake, total tract digestibility, and feeding behavior of beef heifers fed high-concentrate diets. Transl Anim Sci. 2020. [19] Price CJ, Banks PB, Brown S, et al. Invasive mammalian predators habituate to and generalize avian prey cues: a mechanism for conserving native prey. Ecol Appl. 2020. [20] Llonch L, Castillejos L, Ferret A, et al. Increasing the content of physically effective fiber in high-concentrate diets fed to beef heifers affects intake, sorting behavior, time spent ruminating, and rumen pH. J Anim Sci. 2020. [21] Foskolos A, Ferret A, Siurana A, et al. Effects of Capsicum and Propyl-Propane Thiosulfonate on Rumen Fermentation, Digestion, and Milk Production and Composition in Dairy Cows. Animals (Basel). 2020. [22] Fandiño I, Fernandez-Turren G, Ferret A, et al. Exploring Additive, Synergistic or Antagonistic Effects of Natural Plant Extracts on In Vitro Beef Feedlot-Type Rumen Microbial Fermentation Conditions. Animals (Basel). 2020. [23] Calsamiglia S, Espinosa G, Vera G, et al. A virtual dairy herd as a tool to teach dairy production and management. J Dairy Sci. 2020. [24] Salas H, Castillejos L, López-Suárez M, et al. In vitro Digestibility, In situ Degradability, Rumen Fermentation and N Metabolism of Camelina Co-Products for Beef Cattle Studied with A Dual Flow Continuous Culture System of Camelina Co-Products for Beef Cattle. Animals (Basel). 2019. [25] Altman MO, Gagneux P. Absence of Neu5Gc and Presence of Anti-Neu5Gc Antibodies in Humans-An Evolutionary Perspective. Front Immunol. 2019. [26] Madruga A, Abril RS, González LA, et al. Using 19% of alfalfa hay in beef feedlot finishing diets did not modify meat quality but increased feed intake and ADG1. J Anim Sci. 2019. [27] Moya D, Ferret A, Blanch M, et al. Effects of live yeast (Saccharomyces cerevisiae) and type of cereal on rumen microbial fermentation in a dual flow continuous culture fermentation system. J Anim Physiol Anim Nutr (Berl). 2018. [28] Le Bourgot C, Ferret-Bernard S, Apper E, et al. Perinatal short-chain fructooligosaccharides program intestinal microbiota and improve enteroinsular axis function and inflammatory status in high-fat diet-fed adult pigs. FASEB J. 2019. [29] Gallego M. Case Report of a Satin Guinea Pig with Fibrous Osteodystrophy That Resembles Human Pseudohypoparathyroidism. Case Rep Vet Med. 2017. [30] Davis JS, Williams SH. The influence of diet on masticatory motor patterns in musteloid carnivorans: An analysis of jaw adductor activity in ferrets (Mustela putorius furo) and kinkajous (Potos flavus). J Exp Zool A Ecol Integr Physiol. 2017. [31] Blanchard G, Marsot M, Bourassin R, et al. Characterisation of the French ferret population, husbandry, reported medical care and feeding habits. J Nutr Sci. 2018. [32] Madruga A, González LA, Mainau E, et al. Effect of increasing the level of alfalfa hay in finishing beef heifer diets on intake, sorting, and feeding behavior. J Anim Sci. 2018. [33] Madruga A, Mainau E, González LA, et al. Effect of forage source included in total mixed ration on intake, sorting and feeding behavior of growing heifers fed high-concentrate diets. J Anim Sci. 2017. [34] Webb J, Graham J, Fordham M, et al. Diagnosis and treatment of esophageal foreign body or stricture in three ferrets (Mustela putorius furo). J Am Vet Med Assoc. 2017. [35] Madruga A, Mainau E, González LA, et al. Technical note: Recording rules for behavioral studies in growing heifers fed high-concentrate diets. J Anim Sci. 2017. [36] Bakthavatchalu V, Muthupalani S, Marini RP, et al. Endocrinopathy and Aging in Ferrets. Vet Pathol. 2016. [37] Kent LM, Morton DP, Ward EJ, et al. The Influence of Religious Affiliation on Participant Responsiveness to the Complete Health Improvement Program (CHIP) Lifestyle Intervention. J Relig Health. 2016. [38] Redmon JM, Shrestha B, Cerundolo R, et al. Soy isoflavone metabolism in cats compared with other species: urinary metabolite concentrations and glucuronidation by liver microsomes. Xenobiotica. 2016. [39] Kakehi M, Ikenaka Y, Nakayama SM, et al. Uridine Diphosphate-Glucuronosyltransferase (UGT) Xenobiotic Metabolizing Activity and Genetic Evolution in Pinniped Species. Toxicol Sci. 2015. [40] Gargi Y, Levran N, Stein D, et al. Pre-transition Nutrition Dose and Mortality Using a CRP-Free Operational Metabolic Transition Framework: A MIMIC-IV Transportability Analysis. Clin Nutr ESPEN. 2026. [41] Monti N, Kulak M, Lisi C, et al. Stunting, underweight and thinness in internationally adopted children: prevalence and associated factors in a large cohort study. Eur J Pediatr. 2026. [42] Gao H, Liu J, Qu Q, et al. A Mechanism-Guided Delivery System for Long-Acting Postpartum Depression Therapy via Hydrogen-Facilitated Gut Microbiota Reprogramming. ACS Appl Mater Interfaces. 2026. [43] Okanlawon A, Burlen J, Chandrasekhara V, et al. EUS-GUIDED GALLBLADDER DRAINAGE FOR TREATING SYMPTOMATIC CHOLELITHIASIS IN NON-OPERATIVE CANDIDATES WITHOUT CHOLECYSTITIS. Gastrointest Endosc. 2026. [44] Abraham-Aggarwal K, Hung C, Chen X, et al. Does treatment of nonsleepy OSA with CPAP therapy change CVD risk? A systematic review. Sleep Med. 2026. [45] Marchi PH, Príncipe LA, Antonelo DS, et al. Dynamic shifts in the serum lipidome of dogs with obesity before and after weight loss: a pilot study. J Anim Sci. 2026. [46] Coope OC, Spurr TJ, Levington AL, et al. Health Behaviours in Soccer Support Staff: 24-Hour Movement Adherence Is Positively Associated with Diet Quality. Sports (Basel). 2026. [47] Zick A, Schmidt R X, Reynolds C. Assessing food procurement greenhouse gas emissions and food waste in UK fine dining. Clean Food Syst. 2026. [48] Sutini T, Rakhmawati W, Mediani HS, et al. Family Empowerment for Stunting Prevention in Children Aged 0-2 Years: A Concept Analysis. J Multidiscip Healthc. 2026. [49] Wang C, Lv L, Ma R. Prognostic value of the lymphocyte-to-C-reactive protein ratio for mortality in geriatric patients with severe dysphagia requiring artificial nutrition: a retrospective secondary analysis. Front Med (Lausanne). 2026. [50] Wardiman B, Natsir A, Syahrir S, et al. Global research landscape and transfer-oriented synthesis of diet-based enteric methane mitigation in ruminants (2005-2025): A bibliometric and implementation-focused review. Vet World. 2026. [51] Na H, Choi HS, Lee IH, et al. Aggression mediates the association between problematic smartphone use and cyberbullying perpetration: a 6-year longitudinal study of Korean adolescents. Front Public Health. 2026. [52] Zaheer Z. Socio-economic impact of antimicrobial resistance in Pakistan: a comprehensive review. Front Public Health. 2026. [53] Jiang S, Hengudomsub P, Jullamate P. Practical Challenges in Assisting With Mealtimes for People With Dementia: A Qualitative Content Analysis of Caregivers' Experiences. Res Gerontol Nurs. 2026. [54] Llenas-Bladé A, Puig Figueras M, Padilla Padilla C, et al. Weight management in obese cancer patients during curative active treatment: protocol for the CANOBESE feasibility study. Pilot Feasibility Stud. 2026. [55] Nguyen Q, Bridges K, Phuc HK, et al. Intersecting narratives: exploring generational differences in maternal health factors in Vietnamese mothers of children with orofacial clefts. BMC Pregnancy Childbirth. 2026. [56] Jager-Wittenaar H, Sealy M, Barazzoni R, et al. Nutritional screening within the GLIM procedure-Part 1: Development of a conceptual definition of risk of malnutrition using a modified Delphi procedure. Clin Nutr. 2026. [57] Derbala MH, Stephen B, Hwang W, et al. Artificial intelligence-guided analysis of the tumor microenvironment predicts response to pembrolizumab in rare tumors. J Immunother Cancer. 2026. [58] Malik S, Jibrell H, King C, et al. PARENTAL EXPERIENCES WITH SOLD FOOD INTRODUCTION - A QUALITATIVE ANALYSIS OF THE FEEDING APPROACH. Acad Pediatr. 2026. [59] Madsen S, Allender B, Blackford R, et al. Ketogenic diet therapy in pyruvate dehydrogenase deficiency: Global clinical practice from literature and survey data. Mol Genet Metab. 2026. [60] Massawe N, Adam J, Nyangi E, et al. Socioeconomic determinants of minimum dietary diversity among women of reproductive age in Tanzania. Nutrition. 2026. [61] Yang R, Li Y, Sankaran K, et al. Prevalence aware feature selection improves biomarker identification in microbiome studies. Bioinformatics. 2026. [62] Farooq N, Zhang B, Franzisky BL, et al. Assessing plant water status: Part 2 - Non-destructive and remote sensing approaches. J Sci Food Agric. 2026. [63] Crouch SH, Kolkenbeck-Ruh A, Kahn K, et al. Cash transfers targeting adolescent wellbeing: a scoping review of the literature in low- and middle-income countries. Glob Health Action. 2026. [64] Şarahman Kahraman C, Memiç İnan C, Çetiner Ö, et al. The Turkish version of Body Dysmorphic Disorder Screener (BDDS-5): Cross-cultural adaptation and psychometric evaluation. Nutr Health. 2026. [65] Amin A, Haneef A, Altaf S, et al. Crofelemer for chronic noninfectious diarrhea in adults with HIV/AIDS receiving antiretroviral therapy: a review of plant-derived antisecretory pharmacotherapy. Expert Opin Pharmacother. 2026. [66] He Z, Han M, An Y, et al. Development and validation of a machine learning-based model for diagnosing perioperative malnutrition in older adults with hip fracture. Front Nutr. 2026. [67] Gao J, Wu X, Chen Y, et al. Association between oral food processing ability and nutritional status of older adults based on factor analysis. BMC Nutr. 2026. [68] Çetinkaya Ç, Batirel HF. Current Treatment for Mediastinitis. Thorac Surg Clin. 2026. [69] Rondón LJ, Marín R. Biomarkers for assessing magnesium status. Adv Clin Chem. 2026. [70] Jiang J, Wen L, Liu H, et al. The immunomodulatory role of vitamins in tumor: mechanisms and therapeutic implications. Pharmacol Res. 2026. [71] Maetani T, Sugitani T, Tanabe N, et al. Emphysema and airway disease synergistically impair exercise tolerance in smokers: a CT-based study. Respir Physiol Neurobiol. 2026. [72] Vargas C, Bennett R, Needham C, et al. Minimum standards for co-design in public health research: Results from a double consensus process. Public Health. 2026. [73] Jakob ER, Amireault S, Reed JB, et al. Potential intervention targets to promote physical activity among people with multiple sclerosis: A scoping review protocol for evidence of moderation. PLoS One. 2026. [74] Li X, Liu M, Liao A, et al. Dietary Interventions Targeting Maternal Obesity: Intergenerational Effects, Mechanisms, and Translational Insights. Curr Nutr Rep. 2026. [75] Abdul Rahman I, Abu Bakar N, Muhialdin B, et al. Exploring the HNE-inhibitory potential of in silico selected Moringa oleifera defense proteins. J Comput Aided Mol Des. 2026. [76] Van Buren K, Lundstrom EW, Omari A, et al. Self-Reported Mental Health and Desired Workplace Improvements Among Working Parents, United States, 2024. Public Health Rep. 2026. [77] Mohammadi A, Hamidi Esfahani Z, Abolhassani H, et al. ALPS-like Disorder Linked with STAT3 Mutation: De Novo Variant with Bicytopenia and Literature Review. Endocr Metab Immune Disord Drug Targets. 2026. [78] Oluwole OS, Mohd Said F, Daud NFS, et al. Next-generation gluten-free noodles: integration of hydrocolloids, fibers, and bioactive compounds. Food Sci Biotechnol. 2026.