Bacterial Pathogens in Poultry: Prevalence, Public Health Risks, and Control Strategies

Introduction

Poultry production represents a major global protein source, yet bacterial contamination of poultry meat and eggs remains a persistent challenge for veterinary public health. The term "chicken ka bacteria" colloquially refers to the diverse bacterial flora associated with poultry, encompassing both commensal organisms and frank pathogens. Understanding the etiology, epidemiology, and control of these agents is essential for reducing foodborne illness and maintaining flock health. This review addresses the major bacterial pathogens of poultry, their prevalence, zoonotic potential, diagnostic approaches, and integrated control strategies, with emphasis on regulatory frameworks such as FSIS guidelines for Salmonella control.

Etiology of Major Bacterial Pathogens in Poultry

Salmonella enterica

Salmonella enterica is a Gram-negative, facultatively anaerobic rod belonging to the Enterobacteriaceae family. Over 2,500 serovars exist, with host-restricted serovars such as S. Gallinarum and S. Pullorum causing systemic disease in poultry, while broad-host-range serovars including S. Enteritidis and S. Typhimurium are associated with foodborne illness in humans [1, 2]. The question "does all chicken have salmonella" reflects a common misconception; while prevalence is significant, not all poultry carcasses harbor Salmonella. Contamination rates vary by production system, geographic region, and processing stage [3, 4]. Salmonella colonizes the gastrointestinal tract of chickens without necessarily causing clinical disease, leading to fecal shedding and carcass contamination during slaughter [1, 5]. Virulence factors include flagella, fimbriae, type III secretion systems, and lipopolysaccharide, which facilitate adhesion, invasion, and immune evasion [6, 2]. Antimicrobial resistance in Salmonella, including extensively drug-resistant (XDR) strains, has been documented in hatchery environments and dead-in-shell eggs, with class 1 integron gene cassettes playing a key role in resistance dissemination [1].

Campylobacter jejuni and Campylobacter coli

Campylobacter species, particularly C. jejuni and C. coli, are microaerophilic, Gram-negative, spiral-shaped bacteria that are the leading bacterial cause of human gastroenteritis in many developed nations [7, 8, 29]. Poultry are considered the primary reservoir, with high intestinal colonization rates often exceeding 80% in commercial broiler flocks [7, 9]. The question "salmonella chicken only" is misleading, as Campylobacter is equally if not more prevalent in poultry. Campylobacter colonizes the cecal crypts and mucus layer of the chicken intestine without causing disease in the bird, a phenomenon attributed to the absence of specific host receptors or immune evasion mechanisms [8, 9]. Virulence determinants include flagella-mediated motility, adhesion proteins (CadF, FlpA), cytolethal distending toxin (CDT), and lipooligosaccharide [8]. Shedding dynamics in laying hens experimentally infected with C. coli and C. jejuni demonstrate prolonged fecal excretion and internal organ colonization, including the liver and spleen [9].

Avian Pathogenic Escherichia coli (APEC)

Escherichia coli is a Gram-negative, facultatively anaerobic rod that is part of the normal intestinal microbiota of poultry. However, specific pathotypes known as avian pathogenic E. coli (APEC) cause colibacillosis, a significant disease complex in chickens and turkeys [10, 11, 12, 13]. APEC strains possess virulence genes encoding adhesins (F1 and P fimbriae), iron acquisition systems (aerobactin, salmochelin), toxins (hemolysin, cytotoxic necrotizing factor), and protectins (lipopolysaccharide, capsule) [10, 12, 13]. The question "chicken e coli or salmonella" highlights the need to differentiate these two enteric pathogens, as both can contaminate poultry meat and cause human illness. APEC is also a zoonotic concern, as some strains share virulence traits with human extraintestinal pathogenic E. coli (ExPEC) [34]. Quorum sensing mediated by the LuxS/AI-2 system facilitates environmental adaptability and competitive fitness of APEC [10]. The LsrR regulator modulates resistance to oxidative stress by interfering with sulfate assimilation, enhancing survival within the host [13]. Co-infection with H9N2 avian influenza virus promotes APEC adhesion, demonstrating viral-bacterial synergy in respiratory disease [12].

Clostridium perfringens

Clostridium perfringens is a Gram-positive, spore-forming, anaerobic rod that causes necrotic enteritis in poultry, a disease of major economic importance [14, 30]. Type G strains produce NetB toxin, the primary virulence factor for necrotic enteritis, while other toxinotypes produce alpha, beta, epsilon, and iota toxins [14, 30]. Spores persist in litter, feed, and processing environments, contributing to recurrent outbreaks [14]. Biofilm formation by C. perfringens enhances survival on surfaces in meat and poultry processing plants, complicating sanitation efforts [14]. Lytic bacteriophages have been investigated as biocontrol agents against C. perfringens type G, showing in vitro efficacy [30].

Other Bacterial Pathogens

Several other bacteria cause disease in poultry or pose food safety risks. Avibacterium paragallinarum causes infectious coryza, an upper respiratory tract infection in chickens [15, 26]. Mycoplasma gallisepticum and Mycoplasma synoviae are cell-wall-deficient bacteria causing chronic respiratory disease and synovitis, respectively [16, 17]. Arcobacter butzleri, a close relative of Campylobacter, has been isolated from poultry and exhibits resistance to antibiotics and disinfectants [18]. Staphylococcus aureus, including methicillin-resistant S. aureus (MRSA), has been detected in livestock carcasses, including poultry [19]. Coagulase-negative staphylococci from bone lesions in broilers also carry antimicrobial resistance genes [31]. Listeria monocytogenes can contaminate poultry products, with sous vide preservation methods using essential oils showing antibacterial effects [20]. Pseudomonas lundensis is a psychrotrophic spoilage organism that forms biofilms on refrigerated poultry [21]. The question "what is ducks disease" may refer to duck viral enteritis or bacterial infections such as Riemerella anatipestifer infection, though the term is not standardized. Bartonella henselae DNA has been detected in ducks used as blood meal sources for triatomine insects, indicating potential vector-borne transmission [22].

Epidemiology and Prevalence

Prevalence of bacterial pathogens in poultry varies by region, production system, and sampling methodology. Salmonella prevalence in broiler flocks ranges from 5% to 40% depending on serovar and geographic location [3, 2, 4]. Campylobacter colonization rates in broiler chickens often exceed 80% at slaughter age [7, 9, 29]. The question "pathogens is most common in raw poultry meat" is answered by Campylobacter and Salmonella, which are the most frequently reported bacterial causes of foodborne illness linked to poultry consumption. E. coli is nearly ubiquitous on raw poultry carcasses, with the question "e coli on raw chicken" reflecting consumer awareness of this contamination [11, 34]. Clostridium perfringens is commonly isolated from poultry meat and processing environments [14, 30]. Transportation stress increases microbial load on broiler carcasses, with multifactorial assessments linking stress physiology to higher bacterial counts [3]. Longitudinal surveillance of MRSA in livestock carcasses over 12 years in South Korea revealed persistent contamination of poultry products [19]. The question "chicken salmonella uk" reflects regional variation; in the United Kingdom, Salmonella prevalence in poultry has declined due to vaccination and biosecurity programs, though Campylobacter remains a major concern.

Clinical Signs in Poultry

Clinical manifestations of bacterial infections in poultry depend on the pathogen, host age, immune status, and concurrent infections. Salmonella Gallinarum and Pullorum cause fowl typhoid and pullorum disease, respectively, characterized by septicemia, diarrhea, depression, and high mortality in young birds [1, 2]. Paratyphoid Salmonella infections are often subclinical in adult birds but can cause diarrhea and reduced growth in chicks [5]. The question "chicken bacteria disease" encompasses these clinical presentations. Campylobacter does not typically cause disease in chickens, though experimental infections have shown mild intestinal inflammation [9]. APEC causes colibacillosis, presenting as airsacculitis, pericarditis, perihepatitis, salpingitis, and cellulitis [10, 11, 12, 13]. The question "chicken diseases caused by bacteria" includes colibacillosis as a primary example. Clostridium perfringens type G causes necrotic enteritis, with clinical signs including depression, anorexia, diarrhea, and sudden death [14, 30]. Avibacterium paragallinarum causes infectious coryza, with nasal discharge, facial swelling, and conjunctivitis [15, 26]. Mycoplasma gallisepticum infection leads to chronic respiratory disease with rales, coughing, and nasal discharge [17]. The question "chicken neck bacteria" may refer to bacterial cellulitis or abscesses in the neck region, often associated with E. coli or Staphylococcus species.

Public Health Risks and Zoonotic Implications

Poultry-associated bacterial pathogens are a major cause of foodborne illness worldwide. The question "can you get e coli from chicken" is answered affirmatively; E. coli O157:H7 and other Shiga toxin-producing E. coli (STEC) have been linked to poultry, though less frequently than to beef [34]. Extended-spectrum beta-lactamase (ESBL) and carbapenemase-producing enteric pathogens in animal-origin foods, including poultry, represent a growing One Health concern [34]. The question "salmonella chicken baby" highlights the particular vulnerability of infants and young children to salmonellosis, which can cause severe diarrhea, bacteremia, and meningitis. The question "salmonella chicken washing" addresses the dangerous practice of washing raw chicken, which can aerosolize bacteria and contaminate kitchen surfaces. FSIS guidelines advise against washing raw poultry and recommend cooking to an internal temperature of 165 degrees Fahrenheit (74 degrees Celsius) to kill pathogens. The question "cooking chicken kill bacteria" is answered by thermal inactivation kinetics; proper cooking destroys vegetative bacterial cells, though toxins such as those produced by Staphylococcus aureus or Clostridium perfringens may be heat-stable [32]. The question "does cooked chicken grow bacteria" is relevant to post-cooking handling; cooked chicken can be recontaminated and support bacterial growth if not properly stored. The question "reheat chicken kill bacteria" is partially correct; reheating to 165 degrees Fahrenheit kills vegetative cells but may not inactivate preformed toxins. The question "chicken bacteria toxins" refers to enterotoxins produced by S. aureus, C. perfringens, and Bacillus cereus, which can cause food poisoning even after the bacteria are killed [32]. The question "chicken breast bacteria" reflects consumer concern about contamination of specific cuts; bacterial loads vary by product type and processing hygiene. Campylobacteriosis in humans is strongly associated with poultry meat consumption, with genomic studies linking human clinical isolates to broiler chicken meat isolates [29]. The question "salmonella chicken only" is inaccurate; Salmonella is also transmitted through eggs, other meats, produce, and contact with reptiles or pets.

Diagnostic Methods

Diagnosis of bacterial pathogens in poultry relies on culture, molecular detection, and serological methods. Conventional culture for Salmonella involves pre-enrichment in buffered peptone water, selective enrichment in Rappaport-Vassiliadis or tetrathionate broth, and plating on selective agars such as xylose lysine deoxycholate (XLD) or brilliant green agar [1, 4]. Campylobacter isolation requires microaerophilic conditions (5% oxygen, 10% carbon dioxide, 85% nitrogen) and selective media containing antibiotics [7, 9]. E. coli is cultured on MacConkey agar, with further characterization by biochemical tests and serotyping [10, 11]. Clostridium perfringens is cultured anaerobically on blood agar or tryptose sulfite cycloserine (TSC) agar [14, 30].

Molecular methods include polymerase chain reaction (PCR) and real-time PCR for detection of virulence and resistance genes [1, 7, 2, 4]. PMAxx real-time PCR allows differentiation of viable and viable but nonculturable (VBNC) Salmonella in retail meat [4]. Nanopore amplicon sequencing using k-mers to overcome high sequencing error rates enables characterization of complex mixed Salmonella serovar populations [6]. Whole-genome sequencing provides comprehensive genomic insights into lineages, virulence, and antimicrobial resistance profiles of Campylobacter jejuni [8]. Indirect ELISA based on Sptp protein has been developed for detecting Salmonella infection in poultry [5]. Reverse vaccinology approaches have identified candidate antigens for vaccines against Avibacterium paragallinarum [15]. The question "poultry quizlet" may refer to study resources for veterinary students; diagnostic algorithms for bacterial pathogens are commonly included in such materials.

Treatment and Antimicrobial Resistance

Antimicrobial therapy for bacterial infections in poultry is complicated by increasing resistance. Salmonella isolates from poultry show resistance to multiple drug classes, including fluoroquinolones, beta-lactams, and tetracyclines [1, 2]. Campylobacter isolates exhibit resistance to fluoroquinolones and macrolides, which are drugs of choice for human campylobacteriosis [7, 8]. APEC strains carry resistance genes for aminoglycosides, tetracyclines, and sulfonamides [10, 13]. Clostridium perfringens shows resistance to tetracyclines and bacitracin [14, 30]. The question "chicken e coli or salmonella" in the context of treatment underscores the need for accurate diagnosis to guide antimicrobial selection.

Alternatives to conventional antibiotics include bacteriophages, antimicrobial peptides, and phytochemicals. Lytic bacteriophages have shown efficacy against C. perfringens type G [30]. Bacillus-derived antimicrobial peptides offer potential as alternatives to antibiotics in poultry [35]. Thanatin, an Lpt-targeting antimicrobial peptide, has been investigated for sterilization and preservation of foods [23]. Phloretin interacts with Staphylococcus aureus toxin proteins and has been applied to chicken meat for toxin mitigation [32]. Dehydroacetic acid disrupts cold adaptation and biofilm formation in Pseudomonas lundensis, enhancing safety of refrigerated poultry [21]. Paraprobiotics (inactivated probiotic cells) provide stability and safety benefits in broiler production [27]. The synergistic combination of tilmicosin and sinomenine has been explored for Mycoplasma synoviae infection [16]. Herb pair extracts from Ilex rotunda and Cyperus rotundus show preventive effects against avian colibacillosis [11].

Control Strategies

Biosecurity and Management

Biosecurity measures are fundamental to preventing introduction and spread of bacterial pathogens in poultry flocks. These include all-in/all-out production, cleaning and disinfection of facilities, rodent and insect control, and restricted access to poultry houses [24, 14]. Transportation welfare affects microbial load; stress reduction during transport lowers bacterial contamination of carcasses [3]. The question "chicken neck bacteria" may relate to injection site infections or cellulitis, which can be reduced by proper vaccination and injection techniques.

Vaccination

Vaccines are available for Salmonella, E. coli, and Mycoplasma in poultry. Live attenuated and killed Salmonella vaccines reduce colonization and shedding [5]. Reverse vaccinology has identified candidate antigens for Avibacterium paragallinarum [15]. Machine learning insights have been applied to epitope-based and peptide-based vaccines against APEC [28]. Mycoplasma gallisepticum vaccines include live attenuated strains and bacterins [17]. The question "chicken salmonella uk" reflects successful vaccination programs in the UK that have reduced Salmonella prevalence in laying hens.

Processing and Food Safety Interventions

FSIS poultry Salmonella guidelines mandate performance standards for Salmonella reduction in broiler carcasses and ground poultry. Interventions at slaughter include carcass washing, organic acid sprays, and chlorinated water systems [24, 4]. The question "cooking chicken kill bacteria" is addressed by FSIS recommendations for cooking poultry to 165 degrees Fahrenheit. The question "does cooked chicken grow bacteria" highlights the importance of proper cooling and storage; cooked chicken should be refrigerated below 40 degrees Fahrenheit and consumed within 3 to 4 days. The question "reheat chicken kill bacteria" is relevant to leftovers; reheating to 165 degrees Fahrenheit kills vegetative cells. The question "salmonella chicken washing" is addressed by FSIS guidance advising consumers not to wash raw poultry to prevent cross-contamination.

Biofilm Control

Biofilm formation by pathogens including Salmonella, Campylobacter, E. coli, and C. perfringens in processing environments poses a challenge for sanitation [24, 14]. Strategies to mitigate biofilms include enzymatic cleaners, quaternary ammonium compounds, and bacteriophage-based treatments [24, 14, 30]. Dehydroacetic acid has been shown to disrupt biofilm formation in Pseudomonas lundensis [21].

Consumer Education

Public health messaging should address common questions such as "does all chicken have salmonella" (no, but prevalence is significant), "can you get e coli from chicken" (yes), and "chicken bacteria toxins" (some toxins are heat-stable). The question "chicken breast bacteria" can be addressed by emphasizing proper handling and cooking. The question "chicken salmonella uk" reflects regional differences in prevalence and control measures. The question "salmonella chicken baby" underscores the need for vulnerable populations to avoid undercooked poultry. The question "what is ducks disease" should be clarified as potentially referring to duck viral enteritis or bacterial infections such as Riemerella anatipestifer.

Diagnostic and Control Workflow

The following Mermaid diagram illustrates a decision tree for diagnosis and control of bacterial pathogens in poultry.

flowchart TD
    A[Clinical Signs or Routine Surveillance], > B{Specimen Type}
    B, > C[Fecal or Cecal Swab]
    B, > D[Carcass Rinse or Tissue]
    B, > E[Feed or Environmental Sample]
    C, > F[Selective Culture and Isolation]
    D, > F
    E, > F
    F, > G{Presumptive Identification}
    G, > H[Gram Stain and Biochemical Tests]
    G, > I[Molecular Detection PCR/Sequencing]
    I, > J[Virulence and Resistance Gene Profiling]
    H, > J
    J, > K[Antimicrobial Susceptibility Testing]
    K, > L{Pathogen Confirmed}
    L, > M[Report to Veterinary Authority]
    L, > N[Implement Control Measures]
    N, > O[Biosecurity Enhancement]
    N, > P[Vaccination Program]
    N, > Q[Processing Intervention]
    N, > R[Consumer Education]
    O, > S[Monitor and Reassess]
    P, > S
    Q, > S
    R, > S
    S, > A

Conclusion

Bacterial pathogens in poultry, including Salmonella, Campylobacter, E. coli, and Clostridium perfringens, represent significant challenges to animal health and public health. The term "chicken ka bacteria" encompasses these diverse organisms, each with distinct epidemiology, virulence mechanisms, and control requirements. Understanding the prevalence and transmission dynamics of these pathogens is essential for designing effective intervention strategies. Regulatory frameworks such as FSIS poultry Salmonella guidelines provide standards for reducing contamination at the processing level. Consumer education addressing common questions about cooking, washing, and storage of poultry is critical for reducing foodborne illness. Integrated control strategies combining biosecurity, vaccination, antimicrobial stewardship, and processing interventions offer the best approach to mitigating the risks associated with bacterial pathogens in poultry.

References

[1] Eidaroos NH, Khafagy AR, Eldein AE et al. Virulence and Antimicrobial Resistance Gene Profiling of Salmonella Isolated from Dead-in-Shell Eggs and Hatchery Environments with Emphasis on Class 1 Integron Gene Cassette Sequencing in XDR Strains. Microb Pathog. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42331072/

[2] Kingshuk MMR, Alam SB, Rahman MS et al. The Landscape of Salmonella enterica Serovar Gallinarum-Pullorum Antimicrobial Resistance in Bangladesh's Poultry Industry: A Combined Phenotypic and Molecular Study. Microbiologyopen. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42271175/

[3] Fahim MFA, Banik B, Pranto PS et al. Transportation welfare under commercial conditions: A multifactorial assessment of stress physiology, meat quality, and microbial load in broiler chickens. Poult Sci. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42330766/

[4] Pham HT, Nguyen TH, Lam THA et al. Detection of viable and VBNC Salmonella in retail meat using optimized PMAxx real-time PCR. J Microbiol Methods. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42229763/

[5] Xie L, Xia Y, Shen R et al. Establishment of an indirect ELISA method for detecting Salmonella infection based on Sptp protein in poultry. J Microbiol Methods. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42219045/

[6] Ayala-Velastegui D, Mortimer TD, Siceloff AT et al. NanoPop: Nanopore amplicon sequencing of Salmonella virulence genes to characterize complex mixed serovar populations using k-mers to overcome high sequencing error rates. Microb Genom. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42329233/

[7] Abedini Chamgordani S, Rezaei M, Neyriz Naghadehi M. Investigating Campylobacter Contamination of Broiler Chicken in Isfahan Province (Iran) and Evaluating the Antibiotic Resistance Patterns of the Isolates. Vet Med Int. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42327974/

[8] Qumar S, Hamad M, AbuOdeh R et al. Comprehensive genomic insights into lineages, virulence, and antibiotic resistance profiles of Campylobacter jejuni entailing a global One Health scenario. Gut Pathog. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42288821/

[9] Joaquim P, Balbiani F, Socas ML et al. Experimental infection of laying hens with Campylobacter coli and Campylobacter jejuni: Shedding dynamics and internal organ colonization. Comp Immunol Microbiol Infect Dis. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42229090/

[10] Liu C, Hu J, Guo M et al. LuxS facilitates environmental adaptability and competition capability of avian pathogenic Escherichia coli. Poult Sci. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42314259/

[11] Liu H, Yang A, Wei X et al. Preventive effects and underlying mechanisms of Ilex rotunda Thunb.-Cyperus rotundus L. herb pair extract on avian colibacillosis in chickens. Poult Sci. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42308740/

[12] Li Y, Xue Y, Quan Y et al. Direct interaction between avian pathogenic Escherichia coli and H9N2 avian influenza virus promotes bacterial adhesion during their infections. Microbiol Spectr. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42294695/

[13] Kong L, Tu C, Song X et al. Quorum-sensing regulator LsrR modulates resistance to oxidative stress by interfering with sulfate assimilation in avian pathogenic Escherichia coli. J Bacteriol. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42284197/

[14] Chowdhury MAH, Reem CSA, Ashrafudoulla M et al. Biofilm Formation and Spore-Mediated Persistence of Clostridium perfringens in Meat and Poultry Processing Environments and Their Implications for Control Strategies. J Food Sci. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42316807/

[15] Felici ME, Castell SD, Marconi G et al. Identification of candidate antigens for a vaccine against Avibacterium paragallinarum using reverse vaccinology. Poult Sci. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42302611/

[16] Deng B, Wang X, Li M et al. A novel therapeutic strategy for Mycoplasma synoviae infection: Elucidating the synergistic effects and underlying mechanisms of Tilmicosin and Sinomenine. Poult Sci. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42308745/

[17] Sasidharan SK, Elliott KEC, Evans JD et al. Effects of sampling location and frequency on the detection of Mycoplasma gallisepticum populations in the respiratory tract of commercial layer pullets. Poult Sci. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42308738/

[18] Chiarini E, Buzzanca D, Houf K et al. Resistance patterns and gene expression profiles of Arcobacter butzleri under exposure to selected antibiotics and disinfectants. Curr Res Microb Sci. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42328625/

[19] Lee YH, Ali MS, Moon BY et al. Longitudinal surveillance and molecular characterization of methicillin-resistant Staphylococcus aureus (MRSA) from livestock carcasses in South Korea: A 12-year nationwide study (2013-2024). Food Microbiol. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42215217/

[20] Kačániová M, Zhang G, Popovic

[21] Li Y, Lu Y, Zhang Z et al. Dehydroacetic acid disrupts cold adaptation and biofilm formation in Pseudomonas lundensis: A targeted strategy for enhancing microbiological safety of refrigerated poultry. Food Microbiol. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42215219/

[22] Santos LSD, Velho PENF, Oliveira J et al. Between insects and birds: molecular evidence of Bartonella henselae DNA in Rhodnius prolixus (Stål, 1859) from an insectary and Cairina moschata (Linnaeus, 1758) ducks used as triatomine blood meal source. Rev Inst Med Trop Sao Paulo. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42307305/

[23] Xu T, Liu Q, Wu Q et al. Comprehensive investigation of Lpt-targeting antimicrobial peptide Thanatin for sterilization and preservation in foods. Food Res Int. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42270230/

[24] Alsuwat MA, Shah AA, Ullah S et al. Microbial Biofilm Formation to Mitigate Foodborne Pathogens Strategies and Control Measures. Indian J Microbiol. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42325454/

[25] Tabish RW, Lin Y, Rochell SJ et al. Cecal metagenome and mucosal transcriptome of broilers after an enteric challenge and fed diets with different fiber types and concentrations(1). Poult Sci. 2026. URL: https://pubmed.ncbi.nlm.nih.gov/42241759/